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The pooled change in pain intensity standardized mean difference -0. 14, 95 CI -0. 20 to -0. 08 was equivalent to a 3 mm reduction on a 100 mm visual analog scale greater than in the placebo groups. In RCTs, PERs for all-cause adverse events were 81. 2 versus 66. 2; cosat needed to treat to harm 6 95 CI 5 to 8. Finally, no significant impacts were observed on physical or emotional functioning, and low-quality sine-aid ib of improved sleep sine-aid ib overall impression of change was found in patients. Infliximab is a chimeric IgG1 antibody approved for the treatment of Crohns disease, ulcerative colitis, spondyloarthritis, rheumatoid arthritis, psoriatic arthritis, and plaque psoriasis. Evidence shows that infliximab and other tumor necrosis factor TNF inhibitors substantially improve pathological management for all indications.
However, access to such medications varies and is inversely related to the socioeconomic conditions of each country. Patients starting biologic treatment can receive biosimilars such as CT-P13 in many countries. However, the change in individuals who have success with parent infliximab is controversial. Therefore, the objective of the Nor-Switch study led by Guro Lovik from the Department of Rheumatology at Diakonhjemmet Hospital Norway was to examine the switch from original infliximab to the biosimilar CT-P13 considering parameters such as efficacy, safety and immunogenicity.
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